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辐射致癌证据不足,手机用户暂可放心!
 作者: Scott Woolley    时间: 2011年08月01日    来源: 财富中文网
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日本原子弹爆炸幸存者的脑癌发病率往往被用来恐吓手机用户。相比之下,真实情况远没那么可怕。
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    广岛和长崎原子弹爆炸中幸存下来的日本人患上脑瘤,这种事听起来令人心生恐惧,因为它告诉我们癌症可在人体内隐藏数十年,然后突然发生转移。因此,日本的原子弹爆炸为美国的情况提供了一个完美的注解。过去二三十年,尽管美国的手机使用率增长了500倍之巨,但脑癌患者比例却略有下降。

    别着急,英国的《每日邮报》(The Daily Mail)就曾警告手机用户说:“在对广岛原子弹爆炸幸存者进行检查时发现,其癌症病情发展同样缓慢:爆炸10年后,研究人员没发现任何脑癌迹象,但30年后,却发现了大量脑癌患者。”美国一些城市的市政会议和公共无线电广播中的辩论等各种场合,也同样引用了与原子弹爆炸相关的数据。《财富》杂志网站Fortune.com制作的下图显示,在使用手机和被诊断出脑癌之间,根本不存在关联。紧接着,《赫芬顿邮报》(Huffington Post)就发表了一篇文章,引用原子弹爆炸数据对此图进行驳斥。

    Tumors in the brains of Japanese civilians who survived Hiroshima and Nagasaki seem to tell a frightening tale, the story of how a cancer epidemic can remain hidden for several decades and then suddenly metastasize. The atomic bombings thus provide a perfect explanation for why U.S. brain cancer rates have dropped slightly in the past few decades even as Americans' cell phone use rose 500-fold.

    Just wait, Britain's The Daily Mail warned mobile phone users: "the same slow development of problems occurred when the Hiroshima bomb survivors were tested: after ten years researchers found no evidence of brain cancer, but 30 years later many cases were found." Similar evidence from the atomic bombings has been cited in settings as varied as American city council meetings and debates on public radio. After Fortune.com created this chart showing the apparent disconnect between cell phone use and brain cancer diagnoses, an article immediately popped up on the Huffington Post using the A-Bomb evidence to debunk it.

图中文字

绿色:每天美国手机使用总量(分钟/天)

红色:新诊断出的脑癌发病数量(每百万美国人)

    “对于那些深知脑癌存在漫长潜伏期的人而言,虽然目前脑瘤的总体发病率很低,但他们并未因此感到丝毫放松,”德伏拉•戴维斯指出。他写了一本书,论述手机引发的癌症威胁。“日本原子弹爆炸幸存者的脑癌发病率直到第二次世界大战结束40年后才开始增长。”

    如果这些数字属实,那戴维斯的观点必然极具说服力。让手机用户感到庆幸的是,发生在日本的真实事件远非如此。

    戴维斯等人【或者如《镜报》(The Mirror)所持观点:30年后幸存者的脑癌发病率开始增加】:认为第二次世界大战结束40年后,原子弹爆炸幸存者的脑癌发病率方开始增加的说法,从很多层面上说都大错特错。日本地方当局从1958年才开始对肿瘤病人进行登记,因此原子弹爆炸后头13年,根本不存在脑癌发病记录纯属理所当然。而一旦相关数据被集中起来,脑癌的统计数字即迅速呈现出非同寻常的增长。比如说,一项研究【名为《与原子弹爆炸辐射有关的神经系统和脑垂体肿瘤》(Tumors of the Nervous System and Pituitary Gland Associated With Atomic Bomb Radiation Exposure)——译注】对1961年~1974年(即原子弹爆炸后第16年~第29年)间原子弹爆炸幸存者的脑癌发病率进行了调查,发现此间某些幸存者的脑癌发病率增长了5倍。

    该研究的主要作者、辐射致癌研究领域的主要专家戴尔•普利斯顿表示,在他针对原子弹爆炸幸存者的癌症发病率进行的研究中,没有任何迹象显示,“患癌风险突然猛增”。事实上,他猜测说,上世纪50年代,在对身患癌症的原子弹爆炸幸存者进行登记之前,癌症发病率已经开始上升了。而且,他还对将原子弹爆炸辐射与手机辐射相提并论这种做法表示质疑(原因在于原子弹爆炸在瞬间产生巨大电离辐射,而手机只产生上千个小量的电离辐射。)

    而且,位于广岛的辐射效应研究基金会(Radiation Effects Research Foundation)首席科学家表示,就他所知,没有任何迹象显示,第二次世界大战结束 30~40年后,原子弹爆炸幸存者的脑癌发病率突然增加,而且任何时间段内亦不存在此迹象。主要问题在于,幸存者中患脑癌的人比例很低,他认为其中没有模式可寻。辐射效应研究基金会是美日两国政府联合建立的研究项目。该机构自成立以来,一直在审慎地收集并分析与原子弹爆炸幸存者的健康相关的数据。

    原子弹爆炸导致一些人在爆炸后很快患上致命的脑癌这一残酷的事实,恰好与其他辐射引发的癌症的发病模式吻合。尽管不同人患肿瘤的机率各不相同,但由于辐射患上癌症的人却在逐渐增多。此外,原子弹爆炸引致脑癌发病率迅猛增长的看法也与临床辐射治疗中所见的引发肿瘤的模式相抵触:此研究【《脑部遭受大剂量辐射诱发短暂潜伏期脑膜瘤的个案研究与文献分析》(Radiation induced meningioma with a short latent period following high dose cranial irradiation - case report and literature review)——译注】发现,由于辐射引发的脑癌的发病时间因不同人而相差悬殊,平均而言,一般是在遭受辐射后18年左右发病,最短8年,最长28年。

    换言之,如果一个脑癌高发期即将来临的话,科学界会看到“涨潮”的迹象。因此,脑癌发病率未见上升,实际上反而“令人心安”,布朗大学(Brown University)教授、《美国流行病杂志》(Brown University)的前任编辑大卫•萨维兹表示。

    但是,如果要下结论完全否定使用手机与患脑癌之间存在任何联系,还需要再等上一二十年。在此期间,手机使用量会趋于平稳,而脑癌发病率要么保持不变要么继续下降。

    但愿有利数据会越来越多。本期的《生物电磁》(Bio-Electromagnetics)杂志刊发了一篇英格兰脑癌研究报告。该研究发现,“尽管1985年~2003年间手机应用增长迅猛,但1998年~2007年间英格兰脑癌发病率并未出现明显变化。”迄今,对于几十亿手机用户而言,利好消息是:有关辐射引发的脑癌的科学依据显示:(1)如果因辐射引发的脑癌发病率真的大幅上升的话,科学家应该能发现一些明显迹象;(2)迄今为止,他们尚未发现任何相关迹象。

    译者:大海

    "To those who understand the long latencies involved, the absence of a general brain tumor epidemic at this time provides no comfort," wrote Devra Davis, author of a book on cell phones' cancer threat. "Survivors of the atomic bombs that fell on Japan experienced no increase at all in brain cancer until four decades after the war's end."

    It would be a compelling retort, if it were true. Fortunately for cell phone users, the real story from Japan is very different.

    Claims that survivors "experienced no increase at all in brain cancer until four decades after the war's end," as Davis writes (or even just for 30 years, as The Mirror has it) are wrong on multiple levels. Local tumor registries only began in 1958, so of course there are no records of tumors in the first 13 years after the bombing. Once the data began to be collected, an abnormal number of brain tumors quickly became visible. This study, for instance, covers a period from 1961 and 1974 (from 16 to 29 years after the bombing) and found a five-fold increase in brain cancer in some groups of survivors.

    Dale Preston, a leading expert in radiation induced cancers, says there's no sign of a "sudden spike of risk" in any of his research on cancer in bomb survivors. In fact he suspects tumors were already increasing in frequency in the 1950s, before the records were being kept. He is skeptical about drawing parallels between atomic bomb radiation and cell phone radiation (since the A-Bombs deliver ionizing radiation in one big dose, while cell phones deliver non-ionizing radiation in thousands of small doses.)

    And then there's the chief scientist of the Radiation Effects Research Foundation in Hiroshima, who says that he knows of no evidence that's identified a sudden increase in brain cancer after 30 or 40 years, or for that matter any other time frame. The main problem is that so few survivors developed brain tumors that he doesn't see how anyone could suss out any sort pattern. The Radiation Effects foundation is a joint project the American and Japanese governments that has been meticulously accumulating and analyzing data on the surviving civilians' health following the bombings.

    The grim fact that the A-bombs caused some people to get fatal brain cancers soon after exposure fits with the pattern of other radiation-induced cancers, which tend to increase gradually in the affected population, as different people develop tumors at different rates. The idea that the atomic bombs caused brain tumor rates to shoot up in a sudden spike also conflicts with other the tumor-causing pattern seen in medical radiation treatments: this study found huge variation in the onset of radiation-induced brain cancer, estimating an average lag time of 18 years, plus or minus 10 years.

    In other words, if a huge wave of brain cancer is on its way, scientists would expect to see the tide beginning to rise. Thus the lack of any uptick in brain cancers is "comforting," says David Savitz, a Brown University professor and former editor of The American Journal of Epidemiology.

    Still, conclusively disproving the link between cell phones and brain cancer will only come with another decade or two in which phone use plateaus while tumor rates continue to be flat or down.

    Hopefully the evidence will continue to mount. In the current issue of Bio-Electromagnetics a study of brain cancer in England found that "the increased use of mobile phones between 1985 and 2003 has not led to a noticeable change in the incidence of brain cancer in England between 1998 and 2007." For now the very good news for the billions of people who talk on cells phones is that the real scientific evidence on radiation-induced brain cancer indicates that (1) scientists would expect to see some sign of an impending epidemic if one were really on its way and (2) So far they don't.




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